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Another study with CuCl 2 treated Hep G2 cell line demonstrated that mitochondrial toxicity is an intermediate-level event associated with copper toxicity to Hep G2 cells while lysosomal damage is the early event and plasma damage a late feature associated with copper toxicity 34

[22] After absorption into plasma, glutathione needs to be broken down into amino acids and re-synthesized intracellularly

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This landscape illustrates both the promise and challenges: multiple valid targets exist, but translational failures have been common

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