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After treatment with CoCl, BPC157-treated HUVECs failed to form tube-like structures (Supplementary Figure S3B), which is consistent with our analysis that the redox state is a key determinant of BACH1s functional direction

It activates the GIP receptor with approximately equal potency to native GIP, but activates the GLP-1 receptor with roughly five times lower potency than native GLP-1

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[5] Researchers investigating antioxidant mechanisms of other peptides in the longevity cluster may find it useful to cross-reference findings from GHK-Cu molecular mechanisms research, where copper-peptide-mediated antioxidant enzyme upregulation follows a partially convergent pathway