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Other examples of uterine immune stem modulation by trophoblasts are the expression of HLA-G dimers by trophoblasts which induce a tolerogenic phenotype in dendritic cells (DC) (72)
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Additionally, noncoding RNA-circ-ITCH in the exosomes of bone marrow-derived stromal stem cells enhanced the angiogenic capacity of human umbilical vein endothelial cells and accelerated wound healing in mice with diabetic foot ulcers by alleviating ferroptosis under HG conditions [40]
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