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Differential Smad2/3 linker phosphorylation is a crosstalk mechanism of Rho/ROCK and canonical TGF-beta3 signaling in tenogenic differentiation
Overall, these data suggested that mice overexpressing PGC-1 display deleterious effects when challenged with APAP, since they failed in the induction of genes involved in GSH synthesis, thereby limiting the GSH reservoir to contrast acetaminophen overdose
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